Please use the form below to submit comments. Also provide an e-mail address and name. Your e-mail address and/or name will be used only to communicate with you about this or future comments you may submit. I am particularly keen to receive references to published material that contradicts the assertions and arguments I have made.
By submitting the above comment, I grant to Ross Alan Hangartner the right to incorporate the comment in full or in part, literally, paraphrased, or conceptually, as he sees fit, into State of Pain or other writings that he may create in the future. However, I don't grant permission to include my name or e-mail address, or to use them in any other way than to contact me for follow-up. I understand that by submitting the comment I acquire no right of any kind in State of Pain or other writings of Ross Alan Hangartner.
Last updated: Tue, Mar 11, 2025
More rat experiments have discovered links among sympathetic activity, the adrenal glands, and hyperalgesia in somatic nerves. When the emission of epinephrine/norepinephrine by the adrenal glands is chronically increased, it causes general hyperalgesia of somatic sensory nerves.1