Please use the form below to submit comments. Also provide an e-mail address and name. Your e-mail address and/or name will be used only to communicate with you about this or future comments you may submit. I am particularly keen to receive references to published material that contradicts the assertions and arguments I have made.
By submitting the above comment, I grant to Ross Alan Hangartner the right to incorporate the comment in full or in part, literally, paraphrased, or conceptually, as he sees fit, into State of Pain or other writings that he may create in the future. However, I don't grant permission to include my name or e-mail address, or to use them in any other way than to contact me for follow-up. I understand that by submitting the comment I acquire no right of any kind in State of Pain or other writings of Ross Alan Hangartner.
Last updated: Thu, Jun 22, 2017
Secondary punctate hyperalgesia lasts more than twelve hours. The amount of pain from a controlled punctate stimulus is uniform across the area of punctate hyperalgesia and falls off rapidly at the edge. All of this indicates that there is a different mechanism for secondary stroking hyperalgesia than for secondary punctate hyperalgesia.
Secondary punctate hyperalgesia appears to result from increased sensitivity to the input of A-delta fibers. Since A-delta fibers have no heat receptors, this explains why there is no secondary hyperalgesia to heat.